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In renal clear cell carcinoma, HIF2 stimulated the specific enrichment of PUFAs in an ACSL4-dependent manner and promoted the sensitivity to ferroptosis (Zou et al., 2019)

Inflammation (2019) 42(2):42639

Addressing these considerations is essential for integrating H therapy into nephrology practice

GSH depletion and concomitant increase in O&NS and mitochondrial dysfunctions play a role in the pathophysiology of diverse neuroimmune disorders, including depression, myalgic encephalomyelitis/chronic fatigue syndrome and Parkinsons disease, suggesting that depleted GSH is an integral part of these diseases